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IJE Advance Access originally published online on April 17, 2009
International Journal of Epidemiology 2009 38(3):787-790; doi:10.1093/ije/dyp187
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Published by Oxford University Press on behalf of the International Epidemiological Association © The Author 2009; all rights reserved.

Commentary: Understanding the pathophysiology of poverty

F Javier Nieto

Department of Population Health Sciences, School of Medicine and Public Health, University of Wisconsin, Madison, WI, USA.

E-mail: fjnieto@wisc.edu

Accepted 12 March 2009

The first 150 words of the full text of this article appear below.

Simanek et al.'s article in this issue of the Journal1 brings together two fields of research, both of which have a long history and have recently enjoyed a resurgence of interest. The first is the infectious hypothesis of atherosclerosis and the second is the social determinants model for cardiovascular disease (CVD).

The hypothesis that infections might be involved as aetiologic factors in CVD dates back to the late 1800s, though it all but disappeared for most of the 20th century. Interest in his hypothesis, however, surged again during the last two decades of the century, when both experimental and epidemiologic evidence documented its plausibility.2–4 Similarly, even though discussion of the social determinants of health dates back to the very origins of public health,5 newcomers to this field might be surprised to learn that there was a time, not long ago, when ‘social epidemiology’ did not exist as a distinct . . . [Full Text of this Article]


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