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IJE Advance Access originally published online on December 12, 2008
International Journal of Epidemiology 2009 38(1):261-262; doi:10.1093/ije/dyn263
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Published by Oxford University Press on behalf of the International Epidemiological Association © The Author 2008; all rights reserved.

Commentary: Circulating cytokines and risk stratification of stroke incidence—will we do better in future?

Renate B Schnabel and Stefan Blankenberg*

Department of Medicine II, Johannes Gutenberg University-Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

* Corresponding author. Department of Medicine II, Johannes Gutenberg University-Mainz, Langenbeckstr. 1, 55131 Mainz, Germany. E-mail: blankenberg@2-med.klinik.uni-mainz.de

Accepted 10 November 2008

The first 10% of the full text of this article appears below.

Development and complications of atherosclerotic cardiovascular disease are characterized by an elevated inflammatory state. Pro-inflammatory cytokines play a central role in the initiation and perpetuation of inflammatory activity. Since inflammation had been identified as crucial to the atherosclerotic process,1 multiple systemic inflammatory biomarkers have been investigated in relation to outcome in cross-sectional and longitudinal studies. For robust down-stream markers of the inflammatory cascade (i.e. C-reactive protein), positive associations have consistently been demonstrated for coronary endpoints. Stroke, however, is a more diverse phenotype. Confirmed relations of inflammatory markers and incident stroke starting with . . . [Full Text of this Article]


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