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IJE Advance Access originally published online on September 19, 2006
International Journal of Epidemiology 2006 35(5):1355-1356; doi:10.1093/ije/dyl202
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Published by Oxford University Press on behalf of the International Epidemiological Association © The Author 2006; all rights reserved.

Commentary

Commentary: Inflamed about ultrafine particles?

Annette Peters

GSF-National Research Center for Environment and Health, Institute of Epidemiology, Neuherberg, Germany

E-mail: peters@gsf.de

The first 10% of the full text of this article appears below.

Ambient air pollution has been linked to exacerbation of cardiovascular disease morbidity and mortality.1 These associations have been strikingly robust and range from the observation of coronary heart disease exacerbation within hours of exposure to an increased risk of ischaemic heart disease mortality for subjects chronically exposed to elevated concentrations of ambient particles.

Two main pathological processes, atherosclerosis and thrombosis, lead to acute coronary syndromes such as unstable angina and myocardial infarction. The typical atherosclerotic lesion is a fibro-lipid plaque composed of a pool of lipids covered with a connective tissue cap. Although the plaque narrows the coronary arteries, acute coronary . . . [Full Text of this Article]


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