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IJE Advance Access originally published online on January 13, 2005
International Journal of Epidemiology 2005 34(1):52-53; doi:10.1093/ije/dyh393
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IJE vol.34 no.1 © International Epidemiological Association 2005; all rights reserved.

Commentary

Commentary: The risk of variant Creutzfeldt–Jakob Disease: reassurance and uncertainty

RG Will

University of Edinburgh Teviot Place, Edinburgh EH8 9AG, Scotland, UK. E-mail: r.g.will@ed.ac.uk

The first 10% of the full text of this article appears below.

The annual number of deaths from variant Creutzfeldt–Jakob Disease (vCJD) in the UK is currently on a decline.1 Epidemiological and laboratory evidence strongly supports the hypothesis that vCJD is caused by human infection with bovine spongiform encephalopathy (BSE) and the population risk of developing this condition is likely to be proportional to the extent of human exposure to BSE, presumptively through contaminated meat products. The risk of vCJD in countries other than the UK may be due to exposure to indigenous BSE, import of infected animals, animal feed, and food products from the UK, or exposure to BSE during travel to the UK in the risk period 1980–1996. The paper by Chadeau-Hyam and Alperovitch2 assesses these potential risks in France and . . . [Full Text of this Article]


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