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International Journal of Epidemiology 2001;30:1323-1324
© International Epidemiological Association 2001

Perinatal epidemiology

Commentary: Revisiting the primipaternity theory of pre-eclampsia

Brenda Eskenazia and Kim Harleyb

a Center for Children's Environmental Health Research, School of Public Health, University of California, Berkeley, 140 Warren Hall, Berkeley, CA 94720–7380, USA. E-mail: Eskenazi@uclink4.berkeley.edu
b School of Public Health, University of California, Berkeley, 2150 Shattuck Ave., Suite 600, Berkeley, CA 94720–7380, USA. E-mail: kharley@uclink4.berkeley.edu

Pre-eclampsia is characterized by abnormal implantation of the trophoblast in the placenta and poor placental perfusion, followed by sustained hypertension and proteinuria. While the disease has been recognized for centuries, the aetiology of pre-eclampsia is unknown and is likely multifactorial. Clues about the aetiology of pre-eclampsia might be derived from the risk factors that have been identified. However, to date, the only well-accepted risk factor for pre-eclampsia is primiparity. Recently, it has been suggested that primipaternity rather than primiparity is the relevant risk factor.1 That is, that not only are primiparas at high risk but so are multiparas having . . . [Full Text of this Article]

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