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IJE Advance Access originally published online on April 27, 2006
International Journal of Epidemiology 2006 35(3):534-536; doi:10.1093/ije/dyl065
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Published by Oxford University Press on behalf of the International Epidemiological Association © The Author 2006; all rights reserved.

Commentary

Commentary: The analysis of variance and the social complexities of genetic causation

Jeremy Freese1,2

1 Robert Wood Johnson Scholars in Health Policy Research Program, Harvard University, Cambridge, MA, USA
2 Department of Sociology, University of Wisconsin-Madison, Madison, WI, USA

E-mail: jfreese@ssc.wisc.edu

The first 10% of the full text of this article appears below.

Scientific articles published 30 years earlier can be interesting to revisit for various reasons. Lewontin's classic article on the analysis of variance in human behavioural genetics warrants continued attention for perhaps the worst of them: the article makes several correct observations that continue to remain under-appreciated in some research and much discussion about the causal role of genes in human outcomes.1 The lucidity of Lewontin's arguments has historically proven no match for the allure of overly simple characterizations of outcomes as being x% due to genes and (1 – x)% not due to genes.2 Moreover, Lewontin's main points speak beyond questions about genetics and could even be said to prefigure the best parts of more . . . [Full Text of this Article]


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