Skip Navigation


IJE Advance Access originally published online on May 27, 2004
International Journal of Epidemiology 2004 33(5):929-935; doi:10.1093/ije/dyh231
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow All Versions of this Article:
33/5/929    most recent
dyh231v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (10)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Jablonka, E.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Jablonka, E.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

IJE vol.33 no.5 © International Epidemiological Association 2004; all rights reserved.

Reviews

Epigenetic epidemiology

Eva Jablonka

The Cohn Institute for the History and Philosophy of Science and Ideas, Tel-Aviv University, Tel-Aviv 69978, Israel. E-mail: jablonka@post.tau.ac.il

The first 150 words of the full text of this article appear below.

Traditionally, when a disease persists in a population over several generations, it is attributed either to genetic continuity (the inheritance of defective genes) or to environmental continuity (the persistence of adverse conditions or infectious agents). However, during the last two decades, there has been an accumulation of observations that do not slot neatly into either of these categories. It has become clear that the health and general physiology of animals and people can be affected not only by the interplay of their own genes and conditions of life, but also by the inherited effects of the interplay of genes and environment in their ancestors. These ancestral influences on health depend neither on inheriting particular genes, nor on the persistence of the ancestral environment.

The studies that have revealed heritable effects that do not depend on DNA sequence variations have used several different methodologies and had a variety of aims. Some . . . [Full Text of this Article]


    Epigenetic inheritance systems (EIS)
 

    Epigenetic inheritance and human health
 

    Transgenerational epigenetic inheritance
 

    Where do we go from here?
 

    Glossary
 

Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
MutagenesisHome page
Z. Herceg
Epigenetics and cancer: towards an evaluation of the impact of environmental and dietary factors
Mutagenesis, March 1, 2007; 22(2): 91 - 103.
[Abstract] [Full Text] [PDF]


Home page
Int J EpidemiolHome page
E. Jablonka
Commentary: Induction and selection of variations during cancer development
Int. J. Epidemiol., October 1, 2006; 35(5): 1163 - 1165.
[Full Text] [PDF]


Home page
Am J EpidemiolHome page
M. B. Bracken
Genomic Epidemiology of Complex Disease: The Need for an Electronic Evidence-based Approach to Research Synthesis
Am. J. Epidemiol., August 15, 2005; 162(4): 297 - 301.
[Abstract] [Full Text] [PDF]


Home page
Int J EpidemiolHome page
G. Davey Smith
Genetic epidemiology: an 'enlightened narrative'?
Int. J. Epidemiol., October 1, 2004; 33(5): 923 - 924.
[Full Text] [PDF]


Home page
Int J EpidemiolHome page
L. J Palmer
The New Epidemiology: putting the pieces together in complex disease aetiology
Int. J. Epidemiol., October 1, 2004; 33(5): 925 - 928.
[Full Text] [PDF]